If you’ve ever broken a bone, you know firsthand just how horrible traditional casts are. Hell, even if you haven’t broken a bone, you still probably have a decent idea of how awful casts are, assuming you’ve been within smelling distance of anyone who’s wearing one. After about a week they start to smell like a rotting hog carcass that’s been baking in the sun for three days, and often get itchier than a poison oak rash — but thankfully technology might soon make them obsolete.
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3D-printed casts (an idea that’s been around for a couple years now) could alleviate the odor and itch issues caused by plaster casts, but even though they’re not widely available yet,Turkish student Deniz Karasahin has already taken the idea a step further. winner of the 2014 Golden A’Design Award, Karasahin’s Osteoid cast prototype uses tiny ultrasonic vibrations to speed up bone healing time by up to 40 percent.
The bone healing capabilities of low-intensity pulsed ultrasound (LIPUS) have been known for decades, but the treatment is difficult to administer because it requires ultrasound leads to be placed on the skin, directly over the injured area of the bone. With traditional plaster casts this is basically impossible, but a 3D-printed cast that leaves patches of skin open would make it easy. Osteoid’s simple, skeletal design allows ultrasonic drivers to be built directly into the cast.
It’s still just a design prototype at this point, but given the rapid pace at which 3D scanning and printing technologies are progressing, we wouldn’t be surprised to start seeing these kinds of casts adorning the arms of reckless people all over the globe within the next year or two.
1. Talk to someone: Sharing how we feel helps to reduce the inner tension (but make sure it is someone who cares about your feelings).
2. Work on improving your self-esteem: Self-esteem is the way you see and feel about yourself … and there are lots of lots of…
Did you know that:
1. We only remember about 10% of what we read (textbooks, papers, articles etc)
2. We only retain about 20% of what we hear (lectures, talks, podcasts, conversations etc)
3. We only remember about 30% of what we see (videos, graphs and…
PSA - PLEASE READ AND SPREAD HE WORD!!!
IF YOU SEE THIS PLANT AT ALL, DO NOT TOUCH IT!!!
Giant hogweed (Heracleum mantegazzianum) is an invasive herb in the carrot family which was originally brought to North America from Asia and has since become established in the New England, Mid-Atlantic, and Northwest regions of the United States. Giant hogweed grows along streams and rivers and in fields, forests, yards and roadsides, and a giant hogweed plant can reach 14 feet or more in height with compound leaves up to 5 feet in width.Giant Hogweed sap contains toxic chemicals known as Furanocoumarins. When these chemicals come into contact with the skin and are exposed to sunlight, they cause a condition called Phytophotodermatitis, a reddening of the skin often followed by severe blistering and burns. These injuries can last for several months, and even after they have subsided the affected areas of skin can remain sensitive to light for years. Furanocoumarins are also carcinogenic and teratogenic, meaning they can cause cancer and birth defects. The sap can also cause temporary (or even permanent) blindness if introduced into the eyes.
If someone comes into physical contact with Giant Hogweed, the following steps should be taken:
If a reaction occurs, the early application of topical steroids may lessen the severity of the reaction and ease the discomfort. The affected area of skin may remain sensitive to sunlight for a few years, so applying sun block and keeping the affected area shielded from the sun whenever possible are sensible precautions
- Wash the affected area thoroughly with soap and COLD water as soon as possible.
- Keep the exposed area away from sunlight for 48 hours.
- If Hogweed sap gets into the eyes, rinse them with water and wear sunglasses.
- See a doctor if any sign of reaction sets in.PLEASE, DO NOT JUST READ AND SCROLL! THIS IS VERY IMPORTANT AND POTENTIALLY LIFE-SAVING INFORMATION!!!
Extra note: if you live in Oregon, New Jersey, Michigan or New York and see one of these, call your state’s department of agriculture to report it, and trained professionals will come kill it before it can produce seeds and spread.
Frankly, if you see one in general, probably call your DOA and see if there’s a program in place.
Do not burn it, because the smoke will give you the same reaction.
If for some ungodly reason there isn’t a professional who can handle it for you (and please, please use a professional), the DOA of New York has [this guide] for how to deal with it yourself.
OH MY FUCK I HAVE THESE IN MY BACKYARD.
So I searched a bit and found that this was a thing, please be careful! (http://www.dec.ny.gov/animals/39809.html for more info)
Harvard neuroscientists have made a discovery that turns 160 years of neuroanatomy on its head.
Myelin, the electrical insulating material in the body long known to be essential for the fast transmission of impulses along the axons of nerve cells, is not as ubiquitous as thought, according to new work led by Professor Paola Arlotta of the Harvard Stem Cell Institute (HSCI) and the University’s Department of Stem Cell and Regenerative Biology, in collaboration with Professor Jeff Lichtman of Harvard’s Department of Molecular and Cellular Biology.
“Myelin is a relatively recent invention during evolution,” says Arlotta. “It’s thought that myelin allowed the brain to communicate really fast to the far reaches of the body, and that it has endowed the brain with the capacity to compute higher-level functions.”
In fact, loss of myelin is a feature in a number of devastating diseases, including multiple sclerosis and schizophrenia.
But the new research shows that despite myelin’s essential roles in the brain, “some of the most evolved, most complex neurons of the nervous system have less myelin than older, more ancestral ones,” said Arlotta, co-director of the HSCI neuroscience program.
What this means, she said, is that the higher one looks in the cerebral cortex — closer to the top of the brain, which is its most evolved part — the less myelin one finds. Not only that, but “neurons in this part of the brain display a brand-new way of positioning myelin along their axons that has not been previously seen. They have ‘intermittent myelin’ with long axon tracts that lack myelin interspersed among myelin-rich segments.”
“Contrary to the common assumptions that neurons use a universal profile of myelin distribution on their axons, the work indicates that different neurons choose to myelinate their axons differently,” Arlotta said. “In classic neurobiology textbooks, myelin is represented on axons as a sequence of myelinated segments separated by very short nodes that lack myelin. This distribution of myelin was tacitly assumed to be always the same, on every neuron, from the beginning to the end of the axon. This new work finds this not to be the case.”
The results of the research by Arlotta and postdoctoral fellow Giulio Srubek Tomassy, the first author on the report, are published in the latest edition of the journal Science.
The paper is accompanied by a “perspective” by R. Douglas Fields of the Eunice Kennedy Shriver National Institute of Child Health and Human Development at the National Institutes of Health, who said that Arlotta and Tomassy’s findings raise important questions about the purpose of myelin, and “are likely to spark new concepts about how information is transmitted and integrated in the brain.”
Arlotta and Tomassy collaborated closely on the new work with postdoctoral fellow Daniel Berger of the Lichtman lab, which generated one of the two massive electron microscopy databases that made the work possible.
“The fact that it is the most evolved neurons, the ones that have expanded dramatically in humans, suggest that what we’re seeing might be the ‘future.’ As neuronal diversity increases and the brain needs to process more and more complex information, neurons change the way they use myelin to achieve more,” said Arlotta.
Tomassy said it is possible that these profiles of myelination “may be giving neurons an opportunity to branch out and ‘talk’ to neighboring neurons.” For example, because axons cannot make synaptic contacts when they are myelinated, one possibility is that these long myelin gaps may be needed to increase neuronal communication and synchronize responses across different neurons. He and Arlotta postulate that the intermittent myelin may be intended to fine-tune the electrical impulses traveling along the axons, in order to allow the emergence of highly complex neuronal behaviors.